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Finasteride effects on 5α-reductase ferment. This ferment turns testosterone into a more androgen-active dihydrotestosterone (DHT) form. Finasteride is a 5α-reductase inhibitor, in particular, type II and III, and therefore an antiandrogen. It decreases the production of dihydrotestosterone, including in the prostate gland and the scalp. Inhibits the effect of dihydrotestosterone on the development of prostate adenoma. Finasteride decreases the enlargement of the gland, improves urinary flow, and reduces symptoms associated with benign prostatic hypertrophy. It may take several months of treatment to reduce the clinical symptoms of the disease.

Effects of finasteride on 5α-reductase ferment. This ferment transforms testosterone into a form of dihydrotestosterone (DHT) that is more active on androgens. Finasteride is an inhibitor of 5α-reductase, particularly types II and III, and therefore an antiandrogen. It decreases the production of dihydrotestosterone, including in the prostate gland and scalp. Inhibits the effect of dihydrotestosterone on the development of prostatic adenoma. Finasteride decreases gland enlargement, improves urinary flow, and reduces symptoms associated with benign prostatic hypertrophy. Several months of treatment may be required to reduce the clinical symptoms of the disease.