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Finasteride effects on 5α-reductase ferment. This ferment turns testosterone into a more androgen-active dihydrotestosterone (DHT) form. Finasteride is a 5α-reductase inhibitor, in particular, type II and III, and therefore an antiandrogen. It decreases the production of dihydrotestosterone, including in the prostate gland and the scalp. Inhibits the effect of dihydrotestosterone on the development of prostate adenoma. Finasteride decreases the enlargement of the gland, improves urinary flow, and reduces symptoms associated with benign prostatic hypertrophy. It may take several months of treatment to reduce the clinical symptoms of the disease.